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Research progress of nuclear factor-kappa B and alcoholic liver disease
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[1]赵彩彦, 丛斌.核因子-κB与酒精性肝病[J].国外医学.消化系疾病分册, 2002;22 (4) ∶230-232. [2]Diehl AM.Liver disease in alcohol abusers:clinical perspective[J].Alcohol, 2002;27 (1) ∶7. [3]庄辉.酒精性肝病的流行病学[J].中华肝脏病杂志, 2003, 11 (11) ∶698. [4] 韩雪霖, 司英奎.NF-κB的生物学特性及其与肝脏病的关系[J].中国医学杂志, 2007, 5 (1) ∶51-53. [5]Cheng Q, Lee HH, Li Y, et a1.Upregulation of Bcl-x and Bfl-1as a potential mechanism of ehemoresistance, which can be over-come by NF-kappa B inhibition[J].Oncogene, 2000, 19 (42) ∶4936-4940. [6]Wang CY, Guttridge DC, Mayo MW, et a1.NF-kappa B induces expression of Bcl-2homologue A1/Bf-1to preferentially suppress chemotherapy induced apoptosis[J].Mol Cell Biol, 1999, 19 (9) ∶5923-5929. [7]Tumang JR, Hsia CY, Tian W, et a1.IL-6rescues the hypore-sponsi veness of c-Rel deficient B cells independent of Bcl-xl, Mcl-1, and Bcl-2[J].Cell Immunol, 2002, 217 (1-2) ∶47-57. [8]崔香丹, 金武丕, 李香.大鼠酒精性肝病细胞凋亡中Caspase-3、NF-κB的表达[J].陕西医学杂志, 2009, 38 (3) ∶273-275. [9]Hock JB, Pastofino JG.Ethanol, oxidative stress, and cytokine-induced liver cell injury[J].A1cohol, 2002, 27 (1) ∶63-68. [10]Kono H, Rusyn I, Uesugi T, et al.Diphenyleneiodonium sulfate, an NADPHoxidase inhibitor, prevents early alcohol-induced liver injury in the rat[J].Am J Physiol Gastrointest Liver Physiol, 2001, 280 (5) ∶1005-1012. [11]TomZ, Marta K.Oxidative stress and signal transduction path-ways in alcoholic liver disease[J].Clinical and experimental, 2006, 29 (11) ∶110-115. [12]Zamara E, Novo E, Marra F, et al.4-Hydroxynonenal as a selective pro-fibrogenic stimulus for activated human hepatic stellate cells[J].J Hepatol, 2004, 40 (1) ∶60-68. [13]Kai OL, Kalle J, Amin A.Acetaldehyde Prevents Nuclear Factor-Kappa B Activation and Hepatic Inflammation in Ethanol-Fed Rats[J].Laboratroy investigation, 1999, 79 (7) ∶799-806. [14]Fernandez MA, Callejas NA, Casado M, et al.Thioacetamide-in-duced liver regeneration involves the expression of cyclooxygenase2and nitric oxide synthase2in hepatocytes[J].J Hepatol, 2004, 40 (6) ∶963-970. [15]陈科全, 周字.核因子NF-κB与肝纤维化的关系研究现状[J].国际消化病杂志, 2007, 27 (1) ∶9-12. [16]Istvan H, Pranoti M, Arumugam V, et al.The Critical Role of Toll-Like Receptor (TLR) 4in Alcoholic Liver Disease Is Independ-ent of the Common TLR Adapter MyD88[J].Hepatology, 2008, 48 (4) ∶1224-1229. [17]Uesugi T, Froh M, Arteel GE, et al.Toll-like receptor4is in-volved in the mechanism of early alcohol-induced liver injury in mice[J].Hepatology, 2001, 34 (1) ∶101-108. [18]Wheeler MD, Katuna M, Smutney M, et al.Comparison of the effect of adenoviral delivery of three superoxide dismutase genes a-gainst hepatic ischemia-reperfusion injury[J].Human gene thera-py, 2001, 12 (18) ∶2167-2177. [19]Stewart SF, Day CP.The management of alcoholic liver disease[J].J Hepatol, 2003, 38 (1) ∶2-13. [20]范建高, 丁晓东.酒精性肝炎的治疗现状[J].中华肝脏病杂志, 2003, 11 (11) ∶703-704. -
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