肝纤维化中TGF-β/Smad信号通路

徐丽红; 郑勇

肝纤维化中TGF-β/Smad信号通路

徐丽红,
郑勇

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中图分类号: R575.2
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出版历程
- 出版日期: 2006-01-20

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参考文献(17)

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[9] G.J.Inman, F.J.Nicola s, C.S.Hill.Nucleocytoplasmic shuttlingof Smads 2, 3, and 4 permits sensing of TGF-βreceptor activity themolecular mechanisms that control Smad subcellular[M].Cell, 2002, 10∶283-294.

[10]Daisuke Goto, Hiroshi Nakajima, Yumiko Mori, et al.Interaction be-tween Smad Anchor for Receptor Activation and Smad3 Is Not Essentialfor TGF-β/Smad3-Mediated Signaling[J].Biochemical and Biophysi-cal Research Communication, 2001, 281∶1100-1105.

[11] Chenghai Liu, D.A.Marianna, et al.Smads 2 and 3 are differentiallyactivated by transforming growth factor-β (TGF-β) in quiescent andactivated hepatic stellate cells[J].J Biol Chem, 2003, 278∶11721-11728.

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[15]Shigeki Tsukada, John K.Westwick, Kenichi Ikejima, et al.SMADand p38 MAPK signaling pathways independently regulateα1 collagengene expressing in unstimulate and TGF-βstimulated hepatic stellatecells[J].J Biol Chem, 2005, 280∶10055-10064.

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